In 1969, ontdekte Olney letsel aan de hypothalamus, en beschreef een belemmering in de groei, zwaarlijvigheid, en onvruchtbaarheid bij vrouwen. Eveneens werd een groot aantal geobserveerde pathologische veranderingen gevonden in diverse delen van de hersenen die een verbinding hadden met de endocrine functies bij volwassen muizen, die als pasgeborenen monosodium glutamaat hadden gekregen. Langdurige onderzoeken, waaronder onderzoeken over het voeren, waarbij pasgeborenen/baby proefdieren hoeveelheden monosodium glutaminaat (MSG) kregen toegediend en gedurende maanden of jaren geobserveerd werden voordat ze voor hersenonderzoek opgeofferd werden, bevestigden de eerdere bevinding van Dr. Olney. Al in 1980 waren de bewijzen, dat monosodium glutaminaat hersenletsel veroorzaakte zo goed onderbouwd dat onderzoekers die geinteresseerd waren in de werking van de hersenen en/of de ontwikkeling van geneesmiddelen, deze stof bij hun onderzoek gingen gebruiken. Zij begonnen monosodium glutaminaat te gebruiken als een byzonder middel waarmee bepaalde hersencellen konden worden gedood, zodat ziekteverschijnselen waarbij glutaminezuur een rol speelde nader bestudeerd konden worden.

Op dit moment bestaat er grote bezorgdheid over een van de pathologische veranderingen die volgen uit de hersenbeschadigingen die veroorzaakt worden door het gebruik van monosodium glutaminaat, namelijk uitgesproken zwaarlijvigheid. Te veel eten, ongeschikt dieet, junk food, onvoeldoende lichaamsbeweging, psychologische problemen, erfelijkheidsproblemen, en slecht ouderschap worden allemaal aangevoerd als onderliggende oorzaak van uitgesproken zwaarlijvigheid. Maar nooit worden glutaminezuur in monosodium glutaminaat en asparaginezuur dat voorkomt in aspartaam in dit verband genoemd, die er om bekend staan de oorzaak te zijn van zwaarlijvigheid bij proefdieren. Glutaminezuur en asparaginezuur zijn in bouw gelijk die dezelfde receptoren (in het zenuwstelsel) belasten en gelijksoortige hersenbeschadigingen en neuroendocrine stoornissen veroorzaken. Het is ook bekend dat ze elkaars werking versterken.

Er is geen twijfel mogelijk dat verwerkt vrij glutaminezuur (MSG) bij proefdieren hersenletsel veroorzaakt en de daaruit volgende neuroendocrine stoornissen, waaronder zwaarlijvigheid. Onderzoeken hebben aangetoond dat vrij glutaminezuur (en ook verwerkt vrij glutaminezuur [MSG]) de placenta, (A-B) de bloed hersen barriere (BBB) en de vijf circelvormige kanalen, die op zijn minst lekkage vertonen in iedere levensfase, op een ongecontroleerde manier kunnen passeren gedurende de zwangerschap en de ontwikkeling. (C-E) Bovendien wordt de bloed brein barriere gemakkelijk beschadigd door koorts, beroerte, trauma aan het hoofd, attaque's, de opname van verwerkt vrij glutaminezuur, en het normale verouderings proces. (E-F) Het wordt algemeen aangenomen dat vanwege de grotere doorlaatbaarheid van de bloed brein barriere, kinderen in het bijzonder gevaar lopen door de opname van MSG.

Er is geen twijfel mogelijk dat (ook in Nederland) ongeborenen, pasgeborenen, zuigelingen en kinderen, worden bloot gesteld aan steeds groter wordende hoeveelheden MSG.

In de baarmoeder krijgen kinderen al MSG door het dieet van de moeder. (A-B) In de eerste levensmaand krijgen babies MSG door de moedermelk als de moeder MSG gebruikt (dit is nauwelijks te vermijden als moeder kant en klare voeding gebruikt) of door de babyvoeding die allemaal MSG bevatten. De grootste hoeveelheid MSG in babyvoeding bevindt zich in "hypo-allergene" babyvoeding.

Cantani, A; Micera, M.Immunogenicity of hydrolysate formulas in children (part 1). Analysis of 202 reactions. Journal of Investigational Allergology and Clinical Immunology, 2000 Sep-Oct, 10(5):261-76.

Samenvatting: Gehydrolyseerde babyvoeding van koemelkeiwit verscheen in 1940 op de markt met het doel de overgevoeligheid voor koemelk-eiwit te verminderen of teniet te doen. Op dit moment, is de zogenaamde "hypo-allergene" babyvoeding ontwikkeld. Het gebruik van zulke gehydrolyseerde babyvoeding is gebaseerd op de veronderstelling dat vooraf verwerkte eiwitten, als eiwitten en in samenstelling van eiwitten, zorgen voor voedingsstoffen en een niet-antigene vorm. Zodoende wordt gehydrolyseerde babyvoeding geclassificeerd als hypoallergeen. Deze babyvoeding wordt door verhitting en enzymatische hydrolyse verwerkt, zodanig dat de aanpassing en opeenvolgende structuren min of meer worden veranderd. De babyvoeding bevat vooraf verwerkte eiwitten van een minder molekuulgewicht, dan de natuurlijke eiwitbron, waarvan wordt verondersteld dat het het immuunsysteem minder vatbaar maakt.

Gehydrolyseerde babyvoeding blijkt voldoende voeding te bevatten en zuigelingen komen meestal aan in gewicht tot ze de babyvoeding weigeren vanwege de slechte smaak. Er moet echter voorzichtigheid betracht worden wanneer zulke babyvoeding gedurende langere perioden wordt gegeven omdat er geen gegevens bekend zijn over de voedingswaarde bij kinderen die gedurende verschillende maanden uitsluitend gevoed werden met gehydrolyseerde babyvoeding. In dit artikel rapporteren en bespreken we meer dan 202 reacties op verschillende soorten gehydrolyseerde babyvoeding, waaronder gevallen van anaphylactische shock, een levensbedreigende allergische reactie. De reactie tussen verschillende soorten gehydrolyseerde babyvoeding en koemelkeiwitten, en de mogelijkheid van dit soort babyvoeding om een immuun response te veroorzaken zijn besproken. We komen tot de conclusie dan geen van de soorten gehydrolyseerde babyvoeding allergie veroorzaakt, zowel bij allergische kinderen als voor de groep babies met een hoog risico. We stellen bovendien voor dat er dubbel blinde onderzoeken komen die d.m.v. placebo controle de voeding testen bij grote groepen babies, waarbij duidelijk omschreven, geldige diagnostische methoden een betrouwbaarder beeld geven hoe vaak allergie bij gehydrolyseerde babyvoeding voorkomt.

Pasgeboren kinderen krijgen ook te maken met MSG door de inentingen. Alle levende virus vaccins, en sommige, zo niet alle andere, bevatten MSG. Het MSG in vaccins bevind zich kenmerkend in een of meer van de grondstoffen, waarvan niet duidelijk is dat ze MSG bevatten. Tot 1978 kregen kinderen MSG toegediend in die kleine flesjes voeding die we "baby voeding" noemen. Na 10 jaar onder druk zetten van de fabrikanten door bezorgde wetenschappers en ouders, werd MSG "vrijwillig" uit de "babyvoeding" verwijderd. Maar op 7 januari 1998, keurde de EPA (Environmental Protection Agency) het gebruik van verwerkte vrije glutaminezuur (MSG) goed in bestrijdingsmiddelen en groei versterkers (geur en smaak). Fruit, graan, noten, zaden en groenten die met deze producten zijn behandeld kunnen nu in babyvoeding worden gebruikt, zonder bekend te hoeven maken dat het product MSG bevat, of met MSG is behandeld.

Veel snacks (fastfood), en voeding met weinig of geen vet en bewerkte voeding bevatten vrij glutaminezuur (MSG). Soep in blik dat door grote bedrijven wordt verkocht en dingen zoals kant en klare spaghetti of noodles zitten meestal ook stampvol MSG. De dingen die onze kinderen op school kunnen krijgen bij hun lunches zijn zeer twijfelachtig -- ze bevatten waarschijnlijk meer MSG dan de lunches die door de school worden verzorgd.

Sommigen van ons die gevoelig zijn voor de zenuwgif aminozuren: glutaminezuur, asparaginezuur en L-cysteine, vechten al jaren tegen de macht van de glutamaat en aspartaam industrie. Diegenen onder ons die overgevoelig zijn voor bewerkt vrije glutaminezuur (MSG) vragen alleen dat MSG niet verborgen wordt in voeding, geneesmiddelen, cosmetica en dieet producten. We hebben het gevraagd en we hebben gezien hoe onze verzoeken werden behandeld. We hebben gezien hoe de industrie macht heeft over de regering van de VS en hoe de industrie de media beheerst. We weten dat omdat Ajinomoto Co., Inc. de grootste producent is van bewerkt vrij glutaminezuur en het ingredient "monosodium glutaminaat" jaarlijks miljoenen dolars uitgeeft om mensen in de wereld te misleiden, om te geloven dat MSG "veilig" is, zal er niemand in de wetgevende macht zijn, bij de FDA, de Nationale Gezondheidsdienst, of bij het Centrum voor ziekte onderzoek; en er zal geen enkele beambte bij het Amerikaanse Medisch Genootschap, of de Amerikaanse Vereniging van Dietisten of de pers die ook maar een toespeling zal maken dat MSG de oorsprong zou kunnen zijn van zwaarlijvigheid, of er zelfs maar aan zou bijdragen.

Toen deze pagina werd geschreven zochten we toegang via internet tot de Medline database via de Nationale Medische Bibliotheek (www.pubmed.gov). Wij zochten naar onderzoeken met gebruikmaking van de zoekwoorden "zwaarlijvigheid en monosodium glutaminaat," en we kregen 143 verwijzingen (1-143). Ze staan onder dit artikel. U zult opmerken dat de onderzoeken die aangeven dat MSG hersenletsel veroorzaakt en vervolgens zwaarlijvigheid, in het algemeen werden gedaan in de vroege tachtiger jaren. Latere onderzoeken gebruiken glutaminezuur als onderzoek instrument. Samenvattingen van MSG letsel verlagen het lichaamsgewicht van zogende ratten door het verminderen van etmaal verminderen van energie uitgifte en de bijwerkingen na de geboorte van toediening van monosodium glutaminaat op de insuline werking bij volwassen ratten worden hier bij wijze van toelichting toegevoegd. U zult opmerken dat het allemaal dier onderzoeken zijn. In de Verenigde Staten experimenteren we gewoonlijk niet met giftige stoffen op mensen.

• Afvallen en aspartaam
• Depressie en aspartaam
• Depressiviteit volksziekte nr. 1
• kunstmatige zoetstoffen voor jonge kinderen beperken
• Naar de aspartaam website
  
  
  
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[The effects of neonatal monosodium glutamate treatment on sex-odor attractivity and approach behavior in rats] Gaoxiong Yi Xue Ke Xue Za Zhi. 1993 Apr;9(4):232-42. Chinese. PMID: 8320757

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[Effect of perinatal administration of monosodium glutamate (MSG) on the reproductive system of the male rat] Endokrynol Pol. 1993;44(4):497-505. Polish. PMID: 8055818

63: Hotamisligil GS, Shargill NS, Spiegelman BM. Related Articles, Links

Adipose expression of tumor necrosis factor-alpha: direct role in obesity-linked insulin resistance. Science. 1993 Jan 1;259(5091):87-91. PMID: 7678183

64: Leigh FS, Kaufman LN, Young JB. Related Articles, Links

Diminished epinephrine excretion in genetically obese (ob/ob) mice and monosodium glutamate-treated rats. Int J Obes Relat Metab Disord. 1992 Aug;16(8):597-604. PMID: 1382044

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Nitrogen metabolism in obesity induced by monosodium-L-glutamate in rats. Int J Obes Relat Metab Disord. 1992 Aug;16(8):555-64. PMID: 1326485

66: Ochi M, Furukawa H, Yoshioka H, Sawada T, Kusunoki T, Hattori T. Related Articles, Links

Adipocyte dynamics in hypothalamic obese mice during food deprivation and refeeding. J Nutr Sci Vitaminol (Tokyo). 1991 Oct;37(5):479-91. PMID: 1802974

67: Fisher KN, Turner RA, Pineault G, Kleim J, Saari MJ. Related Articles, Links

The postweaning housing environment determines expression of learning deficit associated with neonatal monosodium glutamate (M.S.G.). Neurotoxicol Teratol. 1991 Sep-Oct;13(5):507-13. PMID: 1758404

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Effect of monosodium glutamate treatment during neonatal development on lipogenesis rate and lipoprotein lipase activity in adult rats. Biochem Int. 1991 Jul;24(5):927-35. PMID: 1776958

69: Stanton LW, Ponte PA, Coleman RT, Snyder MA. Related Articles, Links

Expression of CA III in rodent models of obesity. Mol Endocrinol. 1991 Jun;5(6):860-6. PMID: 1922100

70: Maiter D, Underwood LE, Martin JB, Koenig JI. Related Articles, Links

Neonatal treatment with monosodium glutamate: effects of prolonged growth hormone (GH)-releasing hormone deficiency on pulsatile GH secretion and growth in female rats. Endocrinology. 1991 Feb;128(2):1100-6. PMID: 1989848

71: Yoshioka K, Yoshida T, Kondo M. Related Articles, Links

Reduced brown adipose tissue thermogenesis and metabolic rate in pre-obese mice treated with monosodium-L-glutamate. Endocrinol Jpn. 1991 Feb;38(1):75-9. PMID: 1915115

72: Remke H, Wilsdorf A, Rehorek A. Related Articles, Links

Changes of ATPase activities in erythrocytes of rats with hypothalamic obesity. Exp Pathol. 1991;43(1-2):67-73. PMID: 1664343

73: Lowell BB, Flier JS. Related Articles, Links

Differentiation dependent biphasic regulation of adipsin gene expression by insulin and insulin-like growth factor-1 in 3T3-F442A adipocytes. Endocrinology. 1990 Dec;127(6):2898-906. PMID: 2249632

74: Yoshida T, Yoshioka K, Hiraoka N, Kondo M. Related Articles, Links

Effect of nicotine on norepinephrine turnover and thermogenesis in brown adipose tissue and metabolic rate in MSG obese mice. J Nutr Sci Vitaminol (Tokyo). 1990 Apr;36(2):123-30. PMID: 2388096

75: Lowell BB, Napolitano A, Usher P, Dulloo AG, Rosen BS, Spiegelman BM, Flier JS. Related Articles, Links

Reduced adipsin expression in murine obesity: effect of age and treatment with the sympathomimetic-thermogenic drug mixture ephedrine and caffeine. Endocrinology. 1990 Mar;126(3):1514-20. PMID: 2307116

76: Shapiro BH, MacLeod JN, Pampori NA, Morrissey JJ, Lapenson DP, Waxman DJ. Related Articles, Links

Signalling elements in the ultradian rhythm of circulating growth hormone regulating expression of sex-dependent forms of hepatic cytochrome P450. Endocrinology. 1989 Dec;125(6):2935-44. PMID: 2510988

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Obesity-linked regulation of the adipsin gene promoter in transgenic mice. Proc Natl Acad Sci U S A. 1989 Oct;86(19):7490-4. PMID: 2798420

78: Tokuyama K, Himms-Hagen J. Related Articles, Links

Adrenalectomy prevents obesity in glutamate-treated mice. Am J Physiol. 1989 Aug;257(2 Pt 1):E139-44. PMID: 2764098

79: Yoshida T, Yoshioka K, Wakabayashi Y, Kondo M. Related Articles, Links

Effects of cigarette smoke on norepinephrine turnover and thermogenesis in brown adipose tissue in MSG-induced obese mice. Endocrinol Jpn. 1989 Aug;36(4):537-44. PMID: 2583062

80: Brereton IM, Doddrell DM, Oakenfull SM, Moss D, Irving MG. Related Articles, Links

The use of in vivo 2H NMR spectroscopy to investigate the effects of obesity and diabetes mellitus upon lipid metabolism in mice. NMR Biomed. 1989 Jul;2(2):55-60. PMID: 2534903

81: Rosen BS, Cook KS, Yaglom J, Groves DL, Volanakis JE, Damm D, White T, Spiegelman BM. Related Articles, Links

Adipsin and complement factor D activity: an immune-related defect in obesity. Science. 1989 Jun 23;244(4911):1483-7. PMID: 2734615

82: Ahluwalia P, Malik VB. Related Articles, Links

Effects of monosodium glutamate (MSG) on serum lipids, blood glucose and cholesterol in adult male mice. Toxicol Lett. 1989 Feb;45(2-3):195-8. PMID: 2919400

83: Spiegelman BM, Lowell B, Napolitano A, Dubuc P, Barton D, Francke U, Groves DL, Cook KS, Flier JS. Related Articles, Links

Adrenal glucocorticoids regulate adipsin gene expression in genetically obese mice. J Biol Chem. 1989 Jan 25;264(3):1811-5. PMID: 2912985

84: Wilsdorf A, Remke H, Muller F. Related Articles, Links

[Glucose utilization in adipose tissue of rats in chronic somatotropin deficiency] Biomed Biochim Acta. 1989;48(1):51-6. German. PMID: 2673226

85: Yoshioka K, Yoshida T, Kondo M. Related Articles, Links

Effect of acute cold-exposure on norepinephrine turnover and thermogenesis in brown adipose tissue and metabolic rate in MSG-induced obese mice. Jpn J Physiol. 1989;39(6):957-62. PMID: 2632905

86: Ribeiro EB, Marmo MR, Andrade IS, Dolnikoff MS. Related Articles, Links

Effect of fasting on monosodium glutamate-obese rats. Braz J Med Biol Res. 1989;22(7):917-21. PMID: 2629958

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Effects of castration and androgen on glycosuria appearance in the diabetic KK mice induced by monosodium glutamate administration. Nippon Juigaku Zasshi. 1988 Oct;50(5):1121-3. No abstract available. PMID: 3199623

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Regulation of the central nervous system-pituitary-adrenal axis in rats after neonatal treatment with monosodium glutamate. Neuroendocrinology. 1988 Aug;48(2):105-11. PMID: 2851747

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[Studies on the regulation mechanism for sympathetic nervous system activity--using hypothalamic obese mice] Nippon Naibunpi Gakkai Zasshi. 1988 Jul 20;64(7):554-63. Japanese. PMID: 3146514

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Development of the epididymal adipose tissue in monosodium glutamate-induced obese mice. J Nutr Sci Vitaminol (Tokyo). 1988 Jun;34(3):317-26. PMID: 3183780

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Morphology and cell dynamics of adipose tissue in hypothalamic obese mice. Am J Physiol. 1988 May;254(5 Pt 2):R740-5. PMID: 3364603

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Inflammatory edema induced by carrageenin in monosodium glutamate-treated rats. Braz J Med Biol Res. 1988;21(4):837-9. PMID: 3240379

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Development of hypothalamic obesity in growing rats. Exp Pathol. 1988;33(4):223-32. PMID: 3068067

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[Brain development in the symptomatic obesity model mouse with brain dysfunction. II. Dendritic development of neurons in the cerebral cortex] No To Hattatsu. 1987 Nov;19(6):460-9. Japanese. No abstract available. PMID: 3689594

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Severely impaired adipsin expression in genetic and acquired obesity. Science. 1987 Jul 24;237(4813):405-8. PMID: 3299706

96: Lorden JF, Sims JS. Related Articles, Links

Monosodium L-glutamate lesions reduce susceptibility to hypoglycemic feeding and convulsions. Behav Brain Res. 1987 May;24(2):139-46. PMID: 3297094

97: Scallet AC, Faris PL, Beinfeld MC, Olney JW. Related Articles, Links

Hypothalamic neurotoxins alter the content of immunoreactive cholecystokinin in pituitary. Brain Res. 1987 Mar 31;407(2):390-3. PMID: 3567652

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[Brain development of the symptomatic obesity model mouse with brain dysfunction. I. Neuronal proliferation in the cerebellum] No To Hattatsu. 1987;19(4):294-302. Japanese. No abstract available. PMID: 3663404

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Screening of drugs for thermogenic anti-obesity properties: antidepressants. Ann Nutr Metab. 1987;31(2):69-80. PMID: 3592617

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Measurement of tissue catecholamines of obese rats by liquid chromatography and electrochemical detection. Biomed Biochim Acta. 1987;46(11):823-7. PMID: 3446208

101: Lorden JF, Caudle A. Related Articles, Links

Behavioral and endocrinological effects of single injections of monosodium glutamate in the mouse. Neurobehav Toxicol Teratol. 1986 Sep-Oct;8(5):509-19. PMID: 3785512

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Normal levels of hepatic drug-metabolizing enzymes in neonatally induced, growth hormone-deficient adult male and female rats. Drug Metab Dispos. 1986 Sep-Oct;14(5):585-9. PMID: 2876866

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Morphological and cell proliferative study on the growth of visceral organs in monosodium L-glutamate-treated obese mice. J Nutr Sci Vitaminol (Tokyo). 1986 Aug;32(4):395-411. PMID: 3806254

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Monosodium glutamate neurotoxicity: a sex-specific impairment of blood pressure but not vasopressin in developing rats. Brain Res Bull. 1986 Jul;17(1):51-8. PMID: 3756544

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Decreased ketonaemia in the monosodium glutamate-induced obese rats. Life Sci. 1986 Jun 2;38(22):2009-13. PMID: 3713435

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Components of hypothalamic obesity: bipiperidyl-mustard lesions add hyperphagia to monosodium glutamate-induced hyperinsulinemia. Brain Res. 1986 May 28;374(2):380-4. PMID: 3459567

107: Dulloo AG, Miller DS. Related Articles, Links

The effect of parasympathetic drugs on energy expenditure: relevance to the autonomic hypothesis. Can J Physiol Pharmacol. 1986 May;64(5):586-91. PMID: 3730944

108: Dawson R Jr, Callahan MF, Annau Z. Related Articles, Links

Hypothalamic monoamine metabolism in mice: evaluation of drug challenges and neurotoxic insult. Pharmacology. 1986;32(1):25-37. PMID: 2418449

109: Nakajima H, Tochino Y, Fujino-Kurihara H, Yamada K, Gomi M, Tajima K, Kanaya T, Miyazaki A, Miyagawa J, Hanafusa T, et al. Related Articles, Links

Decreased incidence of diabetes mellitus by monosodium glutamate in the non-obese diabetic (NOD) mouse. Res Commun Chem Pathol Pharmacol. 1985 Nov;50(2):251-7. PMID: 3909273

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Defective thermoregulatory thermogenesis in monosodium glutamate-induced obesity in mice. Metabolism. 1985 Jul;34(7):626-30. PMID: 4010523

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Reduced norepinephrine turnover in brown adipose tissue of pre-obese mice treated with monosodium-L-glutamate. Life Sci. 1985 Mar 11;36(10):931-8. PMID: 3974402

112: DePaolo LV, Steger RW. Related Articles, Links

Alterations in immunoreactive somatostatin levels in hypothalamic and gastroenteropancreatic tissue as a consequence of neonatal treatment with monosodium glutamate. Proc Soc Exp Biol Med. 1985 Mar;178(3):419-25. PMID: 2858103

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Reduced norepinephrine turnover in mice with monosodium glutamate-induced obesity. Metabolism. 1984 Nov;33(11):1060-3. PMID: 6493048

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Unimpaired thermogenic response to noradrenaline in genetic (ob/ob) and hypothalamic (MSG) obese mice. Biosci Rep. 1984 Apr;4(4):343-9. PMID: 6733259

115: Frieder B, Grimm VE. Related Articles, Links

Prenatal monosodium glutamate (MSG) treatment given through the mother's diet causes behavioral deficits in rat offspring. Int J Neurosci. 1984 Apr;23(2):117-26. PMID: 6541212

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Plasma lipoproteins of monosodium glutamate-induced obese rats. Int J Obes. 1984;8(5):385-91. PMID: 6394519

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Naloxone-induced suppression of food intake is potentiated by neonatal administration of monosodium glutamate to mice. Neurobehav Toxicol Teratol. 1983 Sep-Oct;5(5):523-6. PMID: 6664410

118: Hirose Y, Ishihara K, Terashi K, Kazumi T, Utsumi M, Morita S, Baba S. Related Articles, Links

[Hypothalamic obesity induced by monosodium glutamate (MSG) in rats: changes in the endocrine pancreas in the course of and after induction obesity] Nippon Naibunpi Gakkai Zasshi. 1983 Feb 20;59(2):196-207. Japanese. No abstract available. PMID: 6133784

119: Barnhart JE, Pizzi WJ. Related Articles, Links

The Monosodium L-glutamate (MSG) syndrome in mice develops independently of housing condition. Neurobehav Toxicol Teratol. 1982 Sep-Oct;4(5):549-56. PMID: 7177307

120: Romagnano MA, Pilcher WH, Bennett-Clarke C, Chafel TL, Joseph SA. Related Articles, Links

Distribution of somatostatin in the mouse brain: effects of neonatal MSG treatment. Brain Res. 1982 Feb 25;234(2):387-98. PMID: 6120749

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Behavioral and neurochemical effects of neonatal administration of monosodium L-glutamate in mice. J Comp Physiol Psychol. 1981 Feb;95(1):71-84. PMID: 7217414

122: Kanarek RB, Marks-Kaufman R. Related Articles, Links

Increased carbohydrate consumption induced by neonatal administration of monosodium glutamate to rats. Neurobehav Toxicol Teratol. 1981 Fall;3(3):343-50. PMID: 7290289

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Diabetic syndrome in the Chinese hamster induced with monosodium glutamate. Experientia. 1980 Feb 15;36(2):232-4. PMID: 6989622

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Juvenile-onset obesity and deficits in caloric regulation in MSG-treated rats. Pharmacol Biochem Behav. 1979 May;10(5):717-21. PMID: 493287

125: Djazayery A, Miller DS, Stock MJ. Related Articles, Links

Energy balances in obese mice. Nutr Metab. 1979;23(5):357-67. PMID: 384301

126: Tanaka K, Shimada M, Nakao K, Kusunoki T. Related Articles, Links

Hypothalamic lesion induced by injection of monosodium glutamate in suckling period and subsequent development of obesity. Exp Neurol. 1978 Oct;62(1):191-9. No abstract available. PMID: 729669

127: Poon TK, Cameron DP. Related Articles, Links

Measurement of oxygen consumption and locomotor activity in monosodium glutamate-induced obesity. Am J Physiol. 1978 May;234(5):E532-4. PMID: 645905

128: Cameron DP, Cutbush L, Opat F. Related Articles, Links

Effects of monosodium glutamate-induced obesity in mice on carbohydrate metabolism in insulin secretion. Clin Exp Pharmacol Physiol. 1978 Jan-Feb;5(1):41-51. PMID: 639358

129: Nemeroff CB, Lipton MA, Kizer JS. Related Articles, Links

Models of neuroendocrine regulation: use of monosodium glutamate as an investigational tool. Dev Neurosci. 1978;1(2):102-9. PMID: 39735

130: Nikoletseas MM. Related Articles, Links

Obesity in exercising, hypophagic rats treated with monosodium glutamate. Physiol Behav. 1977 Dec;19(6):767-73. No abstract available. PMID: 565061

131: Simson EL, Gold RM, Standish LJ, Pellett PL. Related Articles, Links

Axon-sparing brain lesioning technique: the use of monosodium-L-glutamate and other amino acids. Science. 1977 Nov 4;198(4316):515-7. PMID: 910144

132: Djazayery A. Related Articles, Links

Alterations in blood glucose concentrations in obese mice. Proc Nutr Soc. 1977 May;36(1):15A. No abstract available. PMID: 331329

133: Pizzi WJ, Barnhart JE, Fanslow DJ. Related Articles, Links

Monosodium glutamate admlinistration to the newborn reduces reproductive ability in female and male mice. Science. 1977 Apr 22;196(4288):452-4. PMID: 557837

134: Nemeroff CB, Grant LD, Bissette G, Ervin GN, Harrell LE, Prange AJ Jr. Related Articles, Links

Growth, endocrinological and behavioral deficits after monosodium L-glutamate in the neonatal rat: possible involvement of arcuate dopamine neuron damage. Psychoneuroendocrinology. 1977;2(2):179-96. No abstract available. PMID: 601168

135: Cameron DP, Poon TK, Smith GC. Related Articles, Links

Effects of monosodium glutamate administration in the neonatal period on the diabetic syndrome in KK mice. Diabetologia. 1976 Dec;12(6):621-6. PMID: 1001851

136: Pizzi WJ, Barnhart JE. Related Articles, Links

Effects of monosodium glutamate on somatic development, obesity and activity in the mouse. Pharmacol Biochem Behav. 1976 Nov;5(5):551-7. PMID: 1019185

137: Bunyan J, Murrell EA, Shah PP. Related Articles, Links

The induction of obesity in rodents by means of monosodium glutamate. Br J Nutr. 1976 Jan;35(1):25-39. PMID: 1106764

138: Robinzon B, Snapir N, Perek M. Related Articles, Links

The relation between monosodium glutamate inducing brain damage, and body weight, food intake, semen production and endocrine criteria in the fowl. Poult Sci. 1975 Jan;54(1):234-41. PMID: 806066

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Activity increase associated with obesity induced by monosodium glutamate in mice. Am J Physiol. 1973 Oct;225(4):764-5. No abstract available. PMID: 4743367

140: Djazayery A, Miller DS. Related Articles, Links

The use of gold-thioglucose and monosodium glutamate to induce obesity in mice. Proc Nutr Soc. 1973 May;32(1):30A-31A. No abstract available. PMID: 4586579

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Obesity induced by monosodium glutamate in mice. Natl Inst Anim Health Q (Tokyo). 1973 Summer;13(2):91-101. No abstract available. PMID: 4792405

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Effect of monosodium glutamate on some endocrine functions. Neuroendocrinology. 1971;8(3):245-55. No abstract available. PMID: 5093957

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Brain lesions, obesity, and other disturbances in mice treated with monosodium glutamate. Science. 1969 May 9;164(880):719-21. No abstract available. PMID: 5778021